HIV AND HUMAN-COMPLEMENT - MECHANISMS OF INTERACTION AND BIOLOGICAL IMPLICATION

被引：68

作者：

DIERICH, MP

EBENBICHLER, CF

MARSCHANG, P

FUST, G

THIELENS, NM

ARLAUD, GJ

机构：

[1] LUDWIG BOLTZMANN INST AIDS FORSCH,INNSBRUCK,AUSTRIA

[2] LEM,INST BIOL STRUCT,F-38027 GRENOBLE,FRANCE

[3] NATL INST HAEMATOL & BLOOD TRANSFUS,BUDAPEST,HUNGARY

来源：

IMMUNOLOGY TODAY | 1993年 / 14卷 / 09期

关键词：

D O I：

10.1016/0167-5699(93)90246-H

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

Human complement, although not lytic for HIV-1, interacts with the virus and is closely involved in the infectious process. It enhances infection in the absence of antibody, and turns neutralizing,antibodies into agents which increase viral infectivity. In this review M.P. Dierich et al. summarize available information and discuss possible biological implications.

引用

收藏

页码：435 / 440

页数：6

相关论文

共 57 条

[1] CELLULAR PROTEINS BOUND TO IMMUNODEFICIENCY VIRUSES - IMPLICATIONS FOR PATHOGENESIS AND VACCINES [J].

ARTHUR, LO ;

BESS, JW ;

SOWDER, RC ;

BENVENISTE, RE ;

MANN, DL ;

CHERMANN, JC ;

HENDERSON, LE .

SCIENCE, 1992, 258 (5090) :1935-1938

[2] SEPARATION OF SELF FROM NON-SELF IN THE COMPLEMENT-SYSTEM [J].

ATKINSON, JP ;

FARRIES, T .

IMMUNOLOGY TODAY, 1987, 8 (7-8) :212-215

[3] THE AIDS-ASSOCIATED RETROVIRUS IS NOT SENSITIVE TO LYSIS OR INACTIVATION BY HUMAN-SERUM [J].

BANAPOUR, B ;

SERNATINGER, J ;

LEVY, JA .

VIROLOGY, 1986, 152 (01) :268-271

[4] COMPLEMENT RECEPTOR TYPE-2 MEDIATES INFECTION OF THE HUMAN CD4-NEGATIVE RAJI B-CELL LINE WITH OPSONIZED HIV [J].

BOYER, V ;

DELIBRIAS, C ;

NORAZ, N ;

FISCHER, E ;

KAZATCHKINE, MD ;

DESGRANGES, C .

SCANDINAVIAN JOURNAL OF IMMUNOLOGY, 1992, 36 (06) :879-883

[5] COMPLEMENT MEDIATES HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 INFECTION OF A HUMAN T-CELL LINE IN A CD4-INDEPENDENT AND ANTIBODY-INDEPENDENT FASHION [J].

BOYER, V ;

DESGRANGES, C ;

TRABAUD, MA ;

FISCHER, E ;

KAZATCHKINE, MD .

JOURNAL OF EXPERIMENTAL MEDICINE, 1991, 173 (05) :1151-1158

[6] ESTABLISHMENT OF STABLE, CELL-MEDIATED-IMMUNITY THAT MAKES SUSCEPTIBLE MICE RESISTANT TO LEISHMANIA-MAJOR [J].

BRETSCHER, PA ;

WEI, GJ ;

MENON, JN ;

BIELEFELDTOHMANN, H .

SCIENCE, 1992, 257 (5069) :539-542

[7] A T(H)1-]T(H)2 SWITCH IS A CRITICAL STEP IN THE ETIOLOGY OF HIV-INFECTION [J].

CLERICI, M ;

SHEARER, GM .

IMMUNOLOGY TODAY, 1993, 14 (03) :107-110

[8]

Cooper N.R., 1986, IMMUNOBIOLOGY COMPLE, P139

[9] STRUCTURAL AND FUNCTIONAL-RELATIONSHIPS AMONG RECEPTORS AND REGULATORS OF THE COMPLEMENT-SYSTEM [J].

DIERICH, MP ;

SCHULZ, TF ;

EIGENTLER, A ;

HUEMER, H ;

SCHWABLE, W .

MOLECULAR IMMUNOLOGY, 1988, 25 (11) :1043-1051

[10]

DUKOR P, 1974, PROGR IMMUNOLOGY, V11, P99

← 1 2 3 4 5 6 →