蛋白质组在心血管疾病中的研究进展

被引：1

作者：

韦恒

曾志羽

机构：

[1] 广西医科大学第一附属医院心血管研究所

来源：

广西医学 | 2010年 / 32卷 / 04期

关键词：

蛋白质组; 心血管疾病;

D O I：

暂无

中图分类号：

R54 [心脏、血管（循环系）疾病];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：469 / 473

页数：5

共 10 条

[1] Proteomics Technologies and Challenges [J].

William C.S. Cho .

Genomics Proteomics & Bioinformatics, 2007, (02) :77-85

[2] 心肌型脂肪酸结合蛋白用于急性心梗早期诊断的探索性研究 [J].

张鹏 ;

王前 ;

郑磊 ;

曾方银 .

中国实验诊断学, 2007, (01) :63-65

[3] 心功能衰竭大鼠模型心肌线粒体蛋白的差异表达分析 [J].

孙爱军 ;

王克强 ;

李杰 ;

王时俊 ;

樊惠芝 ;

杨原 ;

葛均波 .

中国动脉硬化杂志, 2004, (02) :183-185

[4] Proteomic analysis identifies mitochondrial metabolic enzymes as major discriminators between different stages of the failing human myocardium [J].

Urbonavicius, Sigitas ;

Wiggers, Henrik ;

Botker, Hans Erik ;

Nielsen, Torsten Toftegaard ;

Kimose, Hans Henrik ;

Ostergaard, Morten ;

Lindholt, Jes S. ;

Vorum, Henrik ;

Honore, Bent .

ACTA CARDIOLOGICA, 2009, 64 (04) :511-522

[5]

Right ventricular hypertrophy with early dysfunction: A proteomics study in a neonatal model[J] . Amir M. Sheikh,Cindy Barrett,Nestor Villamizar,Oscar Alzate,Anne Marie Valente,J. Rene’ Herlong,Damian Craig,Andrew Lodge,Jeffrey Lawson,Carmelo Milano,James Jaggers.The Journal of Thoracic and Cardiovascular Surgery . 2009 (5)

[6] Chronic urotensin II infusion enhances macrophage foam cell formation and atherosclerosis in apolipoprotein E-knockout mice [J].

Shiraishi, Yuji ;

Watanabe, Takuya ;

Suguro, Toshiaki ;

Nagashima, Masaharu ;

Kato, Rina ;

Hongo, Shigeki ;

Itabe, Hiroyuki ;

Miyazaki, Akira ;

Hirano, Tsutomu ;

Adachi, Mitsuru .

JOURNAL OF HYPERTENSION, 2008, 26 (10) :1955-1965

[7] Impact of salusin-α and -β on human macrophage foam cell formation and coronary atherosclerosis [J].

Watanabe, Takuya ;

Nishio, Kae ;

Kanome, Tomoko ;

Matsuyama, Taka-Aki ;

Koba, Shinji ;

Sakai, Tetsuo ;

Sato, Kengo ;

Hongo, Shigeki ;

Nose, Kiyoshi ;

Ota, Hidekazu ;

Kobayashi, Youichi ;

Katagiri, Takashi ;

Shichiri, Masayoshi ;

Miyazaki, Akira .

CIRCULATION, 2008, 117 (05) :638-648

[8] Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity [J].

Rajan, Sudarsan ;

Ahmed, Rafeeq P. H. ;

Jagatheesan, Ganapathy ;

Petrashevskaya, Natalia ;

Boivin, Greg P. ;

Urboniene, Dalia ;

Arteaga, Grace M. ;

Wolska, Beata M. ;

Solaro, R. John ;

Liggett, Stephen B. ;

Wieczorek, David F. .

CIRCULATION RESEARCH, 2007, 101 (02) :205-214

[9] Inducible cardiac-restricted expression of enteroviral protease 2A is sufficient to induce dilated cardiomyopathy [J].

Xiong, Dingding ;

Yajima, Toshitaka ;

Lim, Byung-Kwan ;

Stenbit, Antine ;

Dublin, Andrew ;

Dalton, Nancy D. ;

Summers-Torres, Daphne ;

Molkentin, Jeffery D. ;

Duplain, Herve ;

Wessely, Rainer ;

Chen, Ju ;

Knowlton, Kirk U. .

CIRCULATION, 2007, 115 (01) :94-102

[10]

Alterations in mouse cardiac proteome after in vivo myocardial infarction: permanent ischaemia versus ischaemia–reperfusion[J] . TijlDe Celle,FrankVanrobaeys,PeterLijnen,W. MatthijsBlankesteijn,SylviaHeeneman,JozefVan Beeumen,BartDevreese,Jos F. M.Smits,Ben J. A.Janssen.Experimental Physiology . 2005 (4)

← 1 →