Are we making efficient use of clopidogrel?

被引：10

作者：

Kastrati, A

Schömig, A

Schömig, E

机构：

[1] Tech Univ Munich, Med Klinik Rechts Isar 1, Deutsch Herzzentrum, Munich, Germany

[2] Klinikum Univ Koln, Inst Pharmakol, Cologne, Germany

来源：

EUROPEAN HEART JOURNAL | 2004年 / 25卷 / 06期

关键词：

D O I：

10.1016/j.ehj.2004.02.005

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：454 / 456

页数：3

共 17 条

[1]

Caplain H, 1999, SEMIN THROMB HEMOST, V25, P25

[2]

Denninger MH, 1999, SEMIN THROMB HEMOST, V25, P41

[3] Adenosine diphosphate-induced platelet aggregation is associated with P2Y12 gene sequence variations in healthy subjects [J].

Fontana, P ;

Dupont, A ;

Gandrille, S ;

Bachelot-Loza, C ;

Reny, JL ;

Aiach, M ;

Gaussem, P .

CIRCULATION, 2003, 108 (08) :989-995

[4]

GORCHAKOWA O, IN PRESS EUR HEART J

[5] Clopidogrel for coronary stenting - Response variability, drug resistance, and the effect of pretreatment platelet reactivity [J].

Gurbel, PA ;

Bliden, KP ;

Hiatt, BL ;

O'Connor, CM .

CIRCULATION, 2003, 107 (23) :2908-2913

[6]

Harlfinger S, 2004, ANN SCI SESS AM COLL

[7] Identification of the platelet ADP receptor targeted by antithrombotic drugs [J].

Hollopeter, G ;

Jantzen, HM ;

Vincent, D ;

Li, G ;

England, L ;

Ramakrishnan, V ;

Yang, RB ;

Nurden, P ;

Nurden, A ;

Julius, D ;

Conley, PB .

NATURE, 2001, 409 (6817) :202-207

[8] A clinical trial of abciximab in elective percutaneous coronary intervention after pretreatment with clopidogrel [J].

Kastrati, A ;

Mehilli, J ;

Schühlen, H ;

Dirschinger, J ;

Dotzer, F ;

ten Berg, JM ;

Neumann, F ;

Bollwein, H ;

Volmer, C ;

Gawaz, M ;

Berger, PB ;

Schomig, A .

NEW ENGLAND JOURNAL OF MEDICINE, 2004, 350 (03) :232-238

[9] Atorvastatin reduces the ability of clopidogrel to inhibit platelet aggregation - A new drug-drug interaction [J].

Lau, WC ;

Waskell, LA ;

Watkins, PB ;

Neer, CJ ;

Horowitz, K ;

Hopp, AS ;

Tait, AR ;

Carville, DGM ;

Guyer, KE ;

Bates, ER .

CIRCULATION, 2003, 107 (01) :32-37

[10] Contribution of hepatic cytochrome P450 3A4 metabolic activity to the phenomenon of clopidogrel resistance [J].