SOD1 aggregates in ALS: Cause, correlate or consequence?

被引：54

作者：

Brown, RH ^{[1
]}

机构：

[1] Massachusetts Gen Hosp, Day Neuromuscular Res Lab, Charlestown, MA 02129 USA

来源：

NATURE MEDICINE | 1998年 / 4卷 / 12期

关键词：

D O I：

10.1038/3945

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Increased expression of wild-type SOD1 in mice with ALS does not slow disease progression or diminish the pathological changes in motor neurons.

引用

页码：1362 / 1364

页数：3

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