Regulation of back-propagating action potentials in hippocampal neurons

被引：128

作者：

Johnston, D

Hoffman, DA

Colbert, CM

Magee, JC

机构：

[1] Baylor Coll Med, Div Neurosci, Houston, TX 77030 USA

[2] Max Planck Inst Med Res, D-69120 Heidelberg, Germany

[3] Univ Houston, Dept Biol & Biochem, Houston, TX 77204 USA

[4] Louisiana State Univ, Med Ctr, Ctr Neurosci, New Orleans, LA 70112 USA

来源：

CURRENT OPINION IN NEUROBIOLOGY | 1999年 / 9卷 / 03期

关键词：

D O I：

10.1016/S0959-4388(99)80042-7

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

Protein kinase C has recently been shown to modulate the slow recovery from inactivation of Na+ channels in apical dendrites of hippocampal CA1 pyramidal neurons. Moreover, dendritic, A-type K+ channels have been found to be modulated by protein kinases A and C and by mitogen-activated protein kinase. The electrical signalling ability of these dendrites is thus highly regulated by a number of neurotransmitters and second-messenger systems.

引用

页码：288 / 292

页数：5

共 35 条

[1]

Adams J. P., 1997, Society for Neuroscience Abstracts, V23, P1176

[2]

ALZHEIMER C, 1993, J NEUROSCI, V13, P660

[3] FREQUENCY-DEPENDENT PROPAGATION OF SODIUM ACTION-POTENTIALS IN DENDRITES OF HIPPOCAMPAL CA1 PYRAMIDAL NEURONS [J].