Pain

被引：76

作者：

Basbaum, AI ^{[1
]}

Woolf, CJ

机构：

[1] Univ Calif San Francisco, Dept Anat, WM Keck Fdn Ctr Integrat Neurosci, San Francisco, CA 94143 USA

[2] Univ Calif San Francisco, Dept Physiol, San Francisco, CA 94143 USA

[3] Massachusetts Gen Hosp, Dept Anesthesia & Crit Care, Neural Plast Res Grp, Boston, MA 02129 USA

[4] Harvard Univ, Sch Med, Boston, MA 02129 USA

来源：

CURRENT BIOLOGY | 1999年 / 9卷 / 12期

关键词：

D O I：

10.1016/S0960-9822(99)80273-5

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：R429 / R431

页数：3

共 5 条

[1]

Gold MS, 1998, J NEUROSCI, V18, P10345

[2] Preserved acute pain and reduced neuropathic pain in mice lacking PKC gamma [J].

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Chen, C ;

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Basbaum, AI .

SCIENCE, 1997, 278 (5336) :279-283

[3] Tackling pain at the source: New ideas about nociceptors [J].

Snider, WD ;

McMahon, SB .

NEURON, 1998, 20 (04) :629-632

[4] Two sodium channels contribute to the TTX-R sodium current in primary sensory neurons [J].

Tate, S ;

Benn, S ;

Hick, C ;

Trezise, D ;

John, V ;

Mannion, RJ ;

Costigan, M ;

Plumpton, C ;

Grose, D ;

Gladwell, Z ;

Kendall, G ;

Dale, K ;

Bountra, C ;

Woolf, CJ .

NATURE NEUROSCIENCE, 1998, 1 (08) :653-655

[5] The cloned capsaicin receptor integrates multiple pain-producing stimuli [J].

Tominaga, M ;

Caterina, MJ ;

Malmberg, AB ;

Rosen, TA ;

Gilbert, H ;

Skinner, K ;

Raumann, BE ;

Basbaum, AI ;

Julius, D .

NEURON, 1998, 21 (03) :531-543

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