缺血性脑卒中发病机制研究新进展

被引:34
作者
陈东
赵贵德
考宏盛
机构
[1] 大连市中心医院神经外科!
[2] 大连医科大学附属二院神经外科!
关键词
脑卒中; 脑梗塞; 发病机制;
D O I
10.16636/j.cnki.jinn.2001.01.011
中图分类号
R743 [脑血管疾病];
学科分类号
1002 ;
摘要
短暂或持久的局灶性脑缺血可引起一系列病理生理变化导致脑损害 ,且随时间和缺血程度增加而加重。本文就中枢神经系统中兴奋性氨基酸 (谷氨酸 )三种受体 :NMDA、AMPA、metabotropic (促代谢 )受体 ,神经元及神经胶质细胞的去极复极化 ,缺血后的炎症反应及细胞凋亡几方面在缺血性脑卒中中的作用机制作一综述。
引用
收藏
页码:32 / 34
页数:3
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