THE ROLE OF ENDOTHELIAL-CELLS IN INFLAMMATION

被引:787
作者
POBER, JS [1 ]
COTRAN, RS [1 ]
机构
[1] HARVARD UNIV,SCH MED,DEPT PATHOL,BOSTON,MA 02115
关键词
D O I
10.1097/00007890-199010000-00001
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
[No abstract available]
引用
收藏
页码:537 / 544
页数:8
相关论文
共 69 条
[1]   THE SEVERE AND MODERATE PHENOTYPES OF HERITABLE MAC-1, LFA-1 DEFICIENCY - THEIR QUANTITATIVE DEFINITION AND RELATION TO LEUKOCYTE DYSFUNCTION AND CLINICAL-FEATURES [J].
ANDERSON, DC ;
SCHMALSTEIG, FC ;
FINEGOLD, MJ ;
HUGHES, BJ ;
ROTHLEIN, R ;
MILLER, LJ ;
KOHL, S ;
TOSI, MF ;
JACOBS, RL ;
WALDROP, TC ;
GOLDMAN, AS ;
SHEARER, WT ;
SPRINGER, TA .
JOURNAL OF INFECTIOUS DISEASES, 1985, 152 (04) :668-689
[2]   ENDOTHELIAL LEUKOCYTE ADHESION MOLECULE-1 - AN INDUCIBLE RECEPTOR FOR NEUTROPHILS RELATED TO COMPLEMENT REGULATORY PROTEINS AND LECTINS [J].
BEVILACQUA, MP ;
STENGELIN, S ;
GIMBRONE, MA ;
SEED, B .
SCIENCE, 1989, 243 (4895) :1160-1165
[3]   PAF-ACETHER (PLATELET-ACTIVATING FACTOR) INCREASES MICROVASCULAR PERMEABILITY AND AFFECTS ENDOTHELIUM-GRANULOCYTE INTERACTION IN MICROVASCULAR BEDS [J].
BJORK, J ;
LINDBOM, L ;
GERDIN, B ;
SMEDEGARD, G ;
ARFORS, KE ;
BENVENISTE, J .
ACTA PHYSIOLOGICA SCANDINAVICA, 1983, 119 (03) :305-308
[4]   ISOLATION OF NITRIC-OXIDE SYNTHETASE, A CALMODULIN-REQUIRING ENZYME [J].
BREDT, DS ;
SNYDER, SH .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1990, 87 (02) :682-685
[5]   ENDOTHELIUM-DEPENDENT VASCULAR-RESPONSES - MEDIATORS AND MECHANISMS [J].
BRENNER, BM ;
TROY, JL ;
BALLERMANN, BJ .
JOURNAL OF CLINICAL INVESTIGATION, 1989, 84 (05) :1373-1378
[6]   TUMOR NECROSIS FACTOR CACHECTIN INCREASES PERMEABILITY OF ENDOTHELIAL-CELL MONOLAYERS BY A MECHANISM INVOLVING REGULATORY G-PROTEINS [J].
BRETT, J ;
GERLACH, H ;
NAWROTH, P ;
STEINBERG, S ;
GODMAN, G ;
STERN, D .
JOURNAL OF EXPERIMENTAL MEDICINE, 1989, 169 (06) :1977-1991
[7]  
BRIGHAM KL, 1986, AM REV RESPIR DIS, V133, P913
[8]   SYNTHESIS AND RELEASE OF PLATELET-ACTIVATING FACTOR IS INHIBITED BY PLASMA ALPHA-1-PROTEINASE INHIBITOR OR ALPHA-1-ANTICHYMOTRYPSIN AND IS STIMULATED BY PROTEINASES [J].
CAMUSSI, G ;
TETTA, C ;
BUSSOLINO, F ;
BAGLIONI, C .
JOURNAL OF EXPERIMENTAL MEDICINE, 1988, 168 (04) :1293-1306
[9]  
COHNHEIM J, 1889, LECTURES GENERAL PAT, V1
[10]  
COLDITZ I, 1989, AM J PATHOL, V134, P755